Right Heart Cath

Swan-Ganz Hemodynamics Calculator

Swan-Ganz Calculator

Hemodynamic Inputs

Enter measured values

Patient & Lab Data

cm
kg
g/dL
%
%

Systemic Arterial Pressure

mmHg
mmHg
mmHg

PA Catheter

mmHg
mmHg
mmHg
mmHg
mmHg
L/min

Calculated Hemodynamics

Primary display: Fick

Flow and Perfusion

Cardiac OutputCO
--L/min
Reference: 4–6 L/min
Cardiac IndexCI
--L/min/m²
Reference: 2–4 L/min/m²
Venous SaturationSvO₂
--%
Reference: 65–75%
Cardiac Power OutputCPO
--W
Severe LV dysfunction concern below 0.6 W

Filling Pressures

Right Atrial PressureRAP
--mmHg
Reference: 1–6 mmHg
Wedge PressurePCWP
--mmHg
Reference: 6–11 mmHg
RA to PCWP RatioRAP/PCWP
--
Higher values suggest disproportionate right-sided congestion
PA PressureS/D/M
--mmHg
Displayed as systolic/diastolic/mean

Vascular Resistance and RV Function

Systemic Vascular ResistanceSVR
--dyn·s/cm⁵
Reference: 900–1400
Pulmonary Vascular ResistancePVR
--WU
Reference: ≤2 WU · >2 WU is elevated
PA Pulsatility IndexPAPi
--
Severe RV dysfunction concern below 0.9
Transpulmonary GradientTPG
--mmHg
mPAP − PCWP · >12 suggests pulmonary vascular remodeling

Hemodynamic Interpretation

Enter the measured hemodynamic values and select Calculate Hemodynamics.

Swan Interpretation


Hemodynamic Pattern RAP / CVP PCWP CI SVR SvO₂ PVR PAPi RAP/PCWP CPO Typical Interpretation
Low-output / shock profiles
Cardiogenic Shock Biventricular congested low-output profile <2.2 >1400 Normal or ↑ Normal or ↓ Variable <0.6 severe Low forward flow with elevated filling pressures and compensatory vasoconstriction. CI <1.8, CPO <0.6 W, MAP <65, or SvO₂ <60% strengthen the poor-perfusion signal.
LV-Predominant Failure Left-sided congestion Normal or ↑ >15 Normal or ↑ Usually preserved Usually <0.8 Elevated left-sided filling pressure with reduced forward flow and increased LV afterload. Think LV failure when PCWP elevation is disproportionate to RAP.
RV-Predominant Failure Disproportionate right-sided congestion >8 Normal or mildly ↑ Normal or ↑ Often ↑ <0.9 concerning ≥0.8 concerning Normal or ↓ Elevated RAP with relatively lower PCWP suggests disproportionate RV congestion. Low PAPi, elevated RAP/PCWP, and/or elevated PVR strengthen the RV failure pattern.
Mixed Cardiogenic + Distributive Pump failure plus vasodilation Variable Variable <900 Variable Variable Variable Variable Low cardiac output together with low SVR suggests simultaneous pump failure and vasodilatory/distributive physiology.
Hypovolemia / Underfilling Low-preload low-output profile <3 <6 Normal or ↓ Usually preserved Variable Low right- and left-sided filling pressures with low output and compensatory vasoconstriction. Compatible with hypovolemia or excessive preload reduction.
Distributive / Vasodilatory Classic high-output phenotype ↓ or Normal ↓ or Normal >3.5 typical <900; <700 marked Normal or ↑ Normal or ↓ Usually preserved Variable Normal or ↑ High forward flow with reduced systemic vascular resistance. Markedly low SVR strongly supports vasodilatory physiology.
Congestion and pressure patterns
Congested, Preserved Flow Normal Variable Usually preserved Variable Variable Variable Usually preserved Elevated biventricular filling pressures without reduced calculated cardiac index. Represents congestion without a dominant low-output phenotype.
Tamponade Pattern Pressure-equalization clue typically ≥10 typically ≥10 Variable Often ↓ Often ↑ Elevated RAP and PCWP that are close together suggest pressure equalization. RAP and PCWP within 3 mmHg when both are ≥10 mmHg triggers a tamponade clue. Not diagnostic alone.
Pulmonary hypertension patterns
Precapillary PH Variable ≤15 Variable Variable Variable >2 WU May ↓ with RV failure May ↑ with RV failure Variable mPAP >20 mmHg with PCWP ≤15 mmHg and elevated PVR. Supports precapillary pulmonary hypertension physiology.
Isolated Postcapillary PH Normal or ↑ >15 Variable Variable Variable ≤2 WU Usually preserved Variable Variable mPAP >20 mmHg with elevated PCWP but without significantly elevated PVR. Supports isolated postcapillary pulmonary hypertension.
Combined Post + Precapillary PH Normal or ↑ >15 Variable or ↓ Variable Variable >2 WU May ↓ May ↑ Variable mPAP >20 mmHg with elevated PCWP and elevated PVR. Supports combined postcapillary and precapillary pulmonary hypertension.

Swan Basics


CXR Placement


  • Path: SVC → RA → RV → PA → branch
  • Ideal tip: Proximal R/L pulmonary artery, ~3–5 cm beyond carina, near hilum
  • Too proximal: In RV → arrhythmia risk
  • Too distal: In segmental PA → rupture risk

Equations


  • CO (L/min) = (125 × BSA) / [(SaO2 − ScvO2) × 1.36 × 10 × Hgb]
  • CI (L/min/m²) = CO / BSA
  • SVR (dynes·sec/cm^5) = 80 × (MAP − CVP) / CO
  • PVR (Wood units) = (PAmean − PCWP) / CO
  • TPG (mmHg) = mPAP − PCWP
  • Common bedside reference: ≤12 mmHg. Elevated TPG suggests pulmonary pressure beyond passive left-sided pressure transmission, but current PH classification relies on PVR rather than TPG.
  • CPO = (MAP × CO) / 451
  • PAPI = (PAPs − PAPd) / CVP

References

  1. Swan, H. J. C., Ganz, W., Forrester, J., Marcus, H., Diamond, G., & Chonette, D. (1970). Catheterization of the heart in man with use of a flow-directed balloon-tipped catheter. New England Journal of Medicine, 283(9), 447–451. https://doi.org/10.1056/NEJM197008272830902
  2. Forrester, J. S., Diamond, G., Chatterjee, K., & Swan, H. J. C. (1976). Medical therapy of acute myocardial infarction by application of hemodynamic subsets. New England Journal of Medicine, 295(24), 1356–1362. https://doi.org/10.1056/NEJM197612092952406
  3. Stevenson, L. W., & Perloff, J. K. (1989). The limited reliability of physical signs for estimating hemodynamics in chronic heart failure. JAMA, 261(6), 884–888. https://doi.org/10.1001/jama.1989.03420060100040
  4. Nohria, A., Tsang, S. W., Fang, J. C., Lewis, E. F., Jarcho, J. A., Mudge, G. H., & Stevenson, L. W. (2003). Clinical assessment identifies hemodynamic profiles that predict outcomes in patients admitted with heart failure. Journal of the American College of Cardiology, 41(10), 1797–1804. https://doi.org/10.1016/S0735-1097(03)00309-7
  5. Mendoza, D. D., Cooper, H. A., & Panza, J. A. (2007). Cardiac power output predicts mortality across a broad spectrum of patients with acute cardiac disease. American Heart Journal, 153(3), 366–370. https://doi.org/10.1016/j.ahj.2006.11.014
  6. Kang, G., Ha, R., Banerjee, D., & Pulmonary Artery Pulsatility Index Investigators. (2016). Pulmonary artery pulsatility index predicts right ventricular failure after left ventricular assist device implantation. Journal of Heart and Lung Transplantation, 35(1), 67–73. https://doi.org/10.1016/j.healun.2015.06.007
  7. Bertaina, M., Galluzzo, A., Rossello, X., Omedè, P., Montefusco, A., Totaro, S., Bocchino, P. P., Frigo, A. C., Iannaccone, M., De Ferrari, G. M., & D’Ascenzo, F. (2022). Pulmonary artery catheter monitoring in patients with cardiogenic shock: Time for a reappraisal? Cardiac Failure Review, 8, e15. https://doi.org/10.15420/cfr.2021.18
  8. Humbert, M., Kovacs, G., Hoeper, M. M., et al. (2022). 2022 ESC/ERS Guidelines for the diagnosis and treatment of pulmonary hypertension. European Heart Journal, 43(38), 3618–3731. https://doi.org/10.1093/eurheartj/ehac237
  9. Heidenreich, P. A., Bozkurt, B., Aguilar, D., et al. (2022). 2022 AHA/ACC/HFSA Guideline for the Management of Heart Failure. Circulation, 145(18), e895–e1032. https://doi.org/10.1161/CIR.0000000000001063
  10. Naidu, S. S., Baran, D. A., Jentzer, J. C., et al. (2022). SCAI SHOCK Stage Classification Expert Consensus Update. Journal of the Society for Cardiovascular Angiography & Interventions, 1(5), 100008.