Swan-Ganz Calculator
Hemodynamic Inputs
Enter measured valuesPatient & Lab Data
cm
kg
m²
g/dL
%
%
Systemic Arterial Pressure
mmHg
mmHg
mmHg
PA Catheter
mmHg
mmHg
mmHg
mmHg
mmHg
L/min
Calculated Hemodynamics
Primary display: FickFlow and Perfusion
Cardiac OutputCO
--L/min
Reference: 4–6 L/min
Cardiac IndexCI
--L/min/m²
Reference: 2–4 L/min/m²
Venous SaturationSvO₂
--%
Reference: 65–75%
Cardiac Power OutputCPO
--W
Severe LV dysfunction concern below 0.6 W
Filling Pressures
Right Atrial PressureRAP
--mmHg
Reference: 1–6 mmHg
Wedge PressurePCWP
--mmHg
Reference: 6–11 mmHg
RA to PCWP RatioRAP/PCWP
--
Higher values suggest disproportionate right-sided congestion
PA PressureS/D/M
--mmHg
Displayed as systolic/diastolic/mean
Vascular Resistance and RV Function
Systemic Vascular ResistanceSVR
--dyn·s/cm⁵
Reference: 900–1400
Pulmonary Vascular ResistancePVR
--WU
Reference: ≤2 WU · >2 WU is elevated
PA Pulsatility IndexPAPi
--
Severe RV dysfunction concern below 0.9
Transpulmonary GradientTPG
--mmHg
mPAP − PCWP · >12 suggests pulmonary vascular remodeling
Hemodynamic Interpretation
Swan Interpretation
| Hemodynamic Pattern | RAP / CVP | PCWP | CI | SVR | SvO₂ | PVR | PAPi | RAP/PCWP | CPO | Typical Interpretation |
|---|---|---|---|---|---|---|---|---|---|---|
| Low-output / shock profiles | ||||||||||
| Cardiogenic Shock Biventricular congested low-output profile | ↑ | ↑ | ↓ <2.2 | ↑ >1400 | ↓ | Normal or ↑ | Normal or ↓ | Variable | ↓ <0.6 severe | Low forward flow with elevated filling pressures and compensatory vasoconstriction. CI <1.8, CPO <0.6 W, MAP <65, or SvO₂ <60% strengthen the poor-perfusion signal. |
| LV-Predominant Failure Left-sided congestion | Normal or ↑ | ↑ >15 | ↓ | ↑ | ↓ | Normal or ↑ | Usually preserved | Usually <0.8 | ↓ | Elevated left-sided filling pressure with reduced forward flow and increased LV afterload. Think LV failure when PCWP elevation is disproportionate to RAP. |
| RV-Predominant Failure Disproportionate right-sided congestion | ↑ >8 | Normal or mildly ↑ | ↓ | Normal or ↑ | ↓ | Often ↑ | ↓ <0.9 concerning | ↑ ≥0.8 concerning | Normal or ↓ | Elevated RAP with relatively lower PCWP suggests disproportionate RV congestion. Low PAPi, elevated RAP/PCWP, and/or elevated PVR strengthen the RV failure pattern. |
| Mixed Cardiogenic + Distributive Pump failure plus vasodilation | Variable | Variable | ↓ | ↓ <900 | Variable | Variable | Variable | Variable | ↓ | Low cardiac output together with low SVR suggests simultaneous pump failure and vasodilatory/distributive physiology. |
| Hypovolemia / Underfilling Low-preload low-output profile | ↓ <3 | ↓ <6 | ↓ | ↑ | ↓ | Normal or ↓ | Usually preserved | Variable | ↓ | Low right- and left-sided filling pressures with low output and compensatory vasoconstriction. Compatible with hypovolemia or excessive preload reduction. |
| Distributive / Vasodilatory Classic high-output phenotype | ↓ or Normal | ↓ or Normal | ↑ >3.5 typical | ↓ <900; <700 marked | Normal or ↑ | Normal or ↓ | Usually preserved | Variable | Normal or ↑ | High forward flow with reduced systemic vascular resistance. Markedly low SVR strongly supports vasodilatory physiology. |
| Congestion and pressure patterns | ||||||||||
| Congested, Preserved Flow | ↑ | ↑ | Normal | Variable | Usually preserved | Variable | Variable | Variable | Usually preserved | Elevated biventricular filling pressures without reduced calculated cardiac index. Represents congestion without a dominant low-output phenotype. |
| Tamponade Pattern Pressure-equalization clue | ↑ typically ≥10 | ↑ typically ≥10 | ↓ | ↑ | ↓ | Variable | Often ↓ | Often ↑ | ↓ | Elevated RAP and PCWP that are close together suggest pressure equalization. RAP and PCWP within 3 mmHg when both are ≥10 mmHg triggers a tamponade clue. Not diagnostic alone. |
| Pulmonary hypertension patterns | ||||||||||
| Precapillary PH | Variable | ≤15 | Variable | Variable | Variable | ↑ >2 WU | May ↓ with RV failure | May ↑ with RV failure | Variable | mPAP >20 mmHg with PCWP ≤15 mmHg and elevated PVR. Supports precapillary pulmonary hypertension physiology. |
| Isolated Postcapillary PH | Normal or ↑ | ↑ >15 | Variable | Variable | Variable | ≤2 WU | Usually preserved | Variable | Variable | mPAP >20 mmHg with elevated PCWP but without significantly elevated PVR. Supports isolated postcapillary pulmonary hypertension. |
| Combined Post + Precapillary PH | Normal or ↑ | ↑ >15 | Variable or ↓ | Variable | Variable | ↑ >2 WU | May ↓ | May ↑ | Variable | mPAP >20 mmHg with elevated PCWP and elevated PVR. Supports combined postcapillary and precapillary pulmonary hypertension. |
Swan Basics


CXR Placement
- Path: SVC → RA → RV → PA → branch
- Ideal tip: Proximal R/L pulmonary artery, ~3–5 cm beyond carina, near hilum
- Too proximal: In RV → arrhythmia risk
- Too distal: In segmental PA → rupture risk

Equations
Cardiac output (CO)
- CO (L/min) = (125 × BSA) / [(SaO2 − ScvO2) × 1.36 × 10 × Hgb]
Cardiac index (CI)
- CI (L/min/m²) = CO / BSA
Systemic vascular resistance (SVR)
- SVR (dynes·sec/cm^5) = 80 × (MAP − CVP) / CO
Pulmonary vascular resistance (PVR)
- PVR (Wood units) = (PAmean − PCWP) / CO
Transpulmonary gradient (TPG)
- TPG (mmHg) = mPAP − PCWP
- Common bedside reference: ≤12 mmHg. Elevated TPG suggests pulmonary pressure beyond passive left-sided pressure transmission, but current PH classification relies on PVR rather than TPG.
Cardiac power output (CPO)
- CPO = (MAP × CO) / 451
Pulmonary artery pulsatility index (PAPI)
- PAPI = (PAPs − PAPd) / CVP
References
- Swan, H. J. C., Ganz, W., Forrester, J., Marcus, H., Diamond, G., & Chonette, D. (1970). Catheterization of the heart in man with use of a flow-directed balloon-tipped catheter. New England Journal of Medicine, 283(9), 447–451. https://doi.org/10.1056/NEJM197008272830902
- Forrester, J. S., Diamond, G., Chatterjee, K., & Swan, H. J. C. (1976). Medical therapy of acute myocardial infarction by application of hemodynamic subsets. New England Journal of Medicine, 295(24), 1356–1362. https://doi.org/10.1056/NEJM197612092952406
- Stevenson, L. W., & Perloff, J. K. (1989). The limited reliability of physical signs for estimating hemodynamics in chronic heart failure. JAMA, 261(6), 884–888. https://doi.org/10.1001/jama.1989.03420060100040
- Nohria, A., Tsang, S. W., Fang, J. C., Lewis, E. F., Jarcho, J. A., Mudge, G. H., & Stevenson, L. W. (2003). Clinical assessment identifies hemodynamic profiles that predict outcomes in patients admitted with heart failure. Journal of the American College of Cardiology, 41(10), 1797–1804. https://doi.org/10.1016/S0735-1097(03)00309-7
- Mendoza, D. D., Cooper, H. A., & Panza, J. A. (2007). Cardiac power output predicts mortality across a broad spectrum of patients with acute cardiac disease. American Heart Journal, 153(3), 366–370. https://doi.org/10.1016/j.ahj.2006.11.014
- Kang, G., Ha, R., Banerjee, D., & Pulmonary Artery Pulsatility Index Investigators. (2016). Pulmonary artery pulsatility index predicts right ventricular failure after left ventricular assist device implantation. Journal of Heart and Lung Transplantation, 35(1), 67–73. https://doi.org/10.1016/j.healun.2015.06.007
- Bertaina, M., Galluzzo, A., Rossello, X., Omedè, P., Montefusco, A., Totaro, S., Bocchino, P. P., Frigo, A. C., Iannaccone, M., De Ferrari, G. M., & D’Ascenzo, F. (2022). Pulmonary artery catheter monitoring in patients with cardiogenic shock: Time for a reappraisal? Cardiac Failure Review, 8, e15. https://doi.org/10.15420/cfr.2021.18
- Humbert, M., Kovacs, G., Hoeper, M. M., et al. (2022). 2022 ESC/ERS Guidelines for the diagnosis and treatment of pulmonary hypertension. European Heart Journal, 43(38), 3618–3731. https://doi.org/10.1093/eurheartj/ehac237
- Heidenreich, P. A., Bozkurt, B., Aguilar, D., et al. (2022). 2022 AHA/ACC/HFSA Guideline for the Management of Heart Failure. Circulation, 145(18), e895–e1032. https://doi.org/10.1161/CIR.0000000000001063
- Naidu, S. S., Baran, D. A., Jentzer, J. C., et al. (2022). SCAI SHOCK Stage Classification Expert Consensus Update. Journal of the Society for Cardiovascular Angiography & Interventions, 1(5), 100008.